query

Don't laugh at my question but the coding supervisor here wanted me to put this question out to all of you. Is it appropriate to query a physician on a case to clarify CHF after the account has been billed? Pt came in with a-fib with RVR and Chf secondary to a fib. Cardio consult states "diastolic dysfunction" in the IMPRESSION section of consultation. EP physician states "variable degree of LV systolic dysfunction" in History section of Consultation and "Congestive Heart Failure, acute on chronic" in the IMPRESSION section of consultation. Throughout progress notes no other mention of sys or dias is noted but acute chf and acute on chronic CHF is documented. EF documented by both MDs 45-59% This is a chart unfortunately that I initially reviewed, saw the EP MD consult, did not feel query needed query and was unable to get back into chart prior to discharge. Thanks!

Comments

  • edited May 2016
    You are allowed to connect the diastolic and systolic dysfunction to the CHF and you have acute on chronic.

    I do not see that clarification is needed.

    It just needs to be coded correctly and rebilled if it meets the timeframe guidelines.

    Charlene
  • edited May 2016
    We would do the same Charlene
  • I agree, we have had some denials about this, just make sure it was treated.
    Thanks,
    Amy
  • edited May 2016
    The following is an example from the ACDIS Physician Query Boot Camp


    A cause and effect relationship between diagnoses may not be assumed and coded unless documented as such by the attending.

    The patient presented with _____________________________

    Please document the relationship, if any, between ___________ and ___________ in your progress notes and d/c summary.

    I have used this and it has been pretty effective so far.

    Dawn
  • edited May 2016
    Hi Mary,

    My Docs had input into this one and they like it.

    Vanessa Falkoff RN
    Clinical Documentation Coordinator
    University Medical Center
    Las Vegas, NV
    vanessa.falkoff@umcsn.com
    office 702-383-7322
    cell 702-204-0054
  • Excellent Vanessa. Thanks for sharing. Donna
  • edited May 2016
    Hello Mary,

    Maybe this will help


    Karen Maritano, RN
    Clinical Documentation Specialist
    Legacy Health
    Portland, Oregon
    503-413-7154
    kmaritan@lhs.org
  • edited May 2016
    Here are a couple examples from my facility. We have had excellent results with this approach. The formatting will be a little weird here, but the info should make sense..

    In the setting of HTN / CHF/ CKD requiring treatment with IV lasix, are you managing:
    * HHD W/ CKD III/ with acute on chronic DHF
    (Hypertensive heart disease with CKD III with acute on chronic DHF
    * Other- with explanation of the clinical findings
    * Clinically unable to determine
    * Risk Factors:Admits with acute on chronic DHF, HTN, CKD III and A-fib with no Hx of MI.
    * Clinical Indicators:3/16/2013 Echo impressions include, "left ventricular hypertrophy...at least moderate pulmonary hypertension....The left and right atria are severely enlarged"
    * Treatments: Home meds include; Metoprolol, Torsemide, and Amiodarone. Patient was given IV lasix in ER and as in-patient ,Lasix 20 mg IV daily
    Please clarify and document your clinical opinion in the Progress Notes and DS the definitive and/or presumptive diagnosis, related to the above clinical findings. Please include clinical findings supporting your diagnosis.
    ------------------------------------------------------------------------------------------

    In the setting of CHF/HTN/CKD requiring treatment with IV lasix, are you managing:
    * HHD w/ Acute on Chronic SHF and CKD III-IV
    (hypertensive heart Dz w/ Acute on Chronic Systolic HF and CKD III-IV)
    * Other- with explanation of the clinical findings
    * Clinically unable to determine
    * Risk Factors:Admits w/ known a-fib, systolic and valvular heart disease, HTN, and systolic Head failure. Admits w/ acute on chronic SHF.
    * Clinical Indicators: Per review of labs GFR from 2012-2013 fluctuates between 24-35 over last 2 years in carecast. 5/20/2013 ECHO --The left and right atria are severely enlarged, severe pulmonary hypertension, concentric left ventricular hypertrophy, Left ventricular ejection fraction is 45%, & severe tricuspid regurgitation.
    * Treatments: Home meds: Benicar 20 mg 1/2 tablet daily- changed to Benicar to 20 mg daily on admit, Lasix 20 mg daily, & Metoprolol tartrate 50 mg 3 b.i.d. Admits on intravenous Lasix.
    Please clarify and document your clinical opinion in the Progress Notes and DS the definitive and/or presumptive diagnosis, related to the above clinical findings. Please include clinical findings supporting your diagnosis.

    Janice Schoonhoven RN, MSN, CCDS
    Clinical Documentation Integrity
    Manager- PeaceHealth Oregon West Network
  • edited May 2016
    Here is what I use:

    A cause and effect relationship between HTN and Chronic Renal Failure can be assumed per CMS guidelines.
    A cause and effect relationship between HTN and CAD/CHF may not be assumed and coded unless documented as such by the attending physician.
    Patient has a history of HTN, CAD and CKD with presenting diagnosis of CHF.
    Please reply to this query or document in your next progress note the relationship, if any, between HTN and CAD.


    Dawn M. Vitalone, RN, CCDS
    Clinical Documentation Improvement Specialist
    Community Hospital
    219-513-2611
    dvitalone@comhs.org
  • edited May 2016
    A cause and effect relationship between HTN and CKD is assumed per coding guidelines; but a cause and effect relationship between HTN and CAD may not be assumed unless documented as such by the attending physician.

    Patient has a history of HTN, CAD and CKD and presented with acute CHF.

    Please reply to this query the relationship, if any, between HTN and CAD. (see below)

    *Hypertensive heart disease
    *Hypertensive kidney disease
    *Hypertensive heart & kidney disease
    *Heart disease only
    *Other (please specify)
    *Unknown (please state rationale)

    ls




  • Dr. G:



    Would this statement be congruent or consistent with potential hypertensive CHF with diastolic dysfunction?





    I look for LVH, normal LVEJ% and diastolic dysfunction. I find this more often in women, than in men. I noted that Digoxin is not preferred in such patients as it increases the pumping action of the heart, which is not desired to treat diastolic CHF.



    **************************************************************************************************************************************************************************************

    Available data indicate that brain natriuretic peptide levels are not as high in diastolic heart failure as they are in systolic heart failure. The diagnosis of diastolic heart failure can be made on the basis of left ventricular hypertrophy, clinical evidence of heart failure, and a normal ejection fraction, as well as Doppler findings that are consistent with diastolic dysfunction and elevated filling pressures. The initial treatment of diastolic heart failure should be directed at reducing the congestive state (with the use of diuretics). Long-term goals are to control congestion and to eliminate or reduce the factors, including hypertension, tachycardia, and ischemia, that confer a predisposition to diastolic dysfunction.



    DHF cannot usually be distinguished from SHF by patient history, physical exam, x-ray, and EKG alone. Diagnosis requires an estimate of LV size and EF. These measurements can be made using echo, MUGA, or catheterization. Really, DHF diagnosis is a matter of ruling out other possible causes in patients seeming to have heart failure but who have normal heart size and EF.





    [cid:image004.jpg@01D1752E.F538F0B0]






    (Note that a thickened LV wall (hypertrophy) in a patient with an LVEF >40% is one criteria for the diagnosis of diastolic CHF)



    [cid:image003.png@01D1752E.F2EAE200]



    Paul Evans, RHIA, CCS, CCS-P, CCDS



    Manager, Regional Clinical Documentation

    Sutter West Bay

    633 Folsom St., 7th Floor, Office 7-044

    San Francisco, CA 94107

    Cell: 415.412.9421







    evanspx@sutterhealth.org








  • As always, Dr. G., thank you for the valuable feedback.

    Paul

    Paul Evans, RHIA, CCS, CCS-P, CCDS

    Manager, Regional Clinical Documentation
    Sutter West Bay
    633 Folsom St., 7th Floor, Office 7-044
    San Francisco, CA 94107
    Cell: 415.412.9421



    evanspx@sutterhealth.org


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